Electrocardiografia
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![Page 1: Electrocardiografia](https://reader034.fdocuments.co/reader034/viewer/2022051400/559ef0721a28ab54438b47f9/html5/thumbnails/1.jpg)
ELECTROCARDIOGRAFIA
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1) VELOCIDAD DE 50 MM/SEG
2) DERIVACION DE LEWIS
3) OBSERVAR SEGMENTO ST Y ONDA T
4) HACER DERIVACIONES ESOFAGICAS
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ESTUDIO ELECROFISIOLOGICO
EKG
TEST DE ESFUERZO HOLTER
HISTORIA CLINICANADA PUEDE
REMPLAZARLA
EXAMINA BIEN A TU
PACIENTE
ESCUCHA AL PACIENTE
QUE EL TE SOPLA EL DX
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HC
MONITOR CDE
EKG-VIA VENOSA-LABORATORIO
FALLA HEMODINAMICA
CARDIOVERSION ELECTRICA
monofásico O BIFASICO
100-200-300 JOULES
DX
ESTABILIDAD HEMODINAMICA
DX
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ARRITMIAS
DEL TEJIDO DE LA
UNION AV
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Se presenta en los trastornos de conducción de los impulsos
sinusales como en la bradicardia sinusal paro sinusal o bloqueo
sinusal falla el marcapaso principal (seno) y toma el comando del
corazón el nodo como marcapaso secundario manejando frecuencias cardiacas de 40 a 60 por minuto
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• PRECEDE AL QRS , NEGATIVA DII-DIII-VF
POSITIVA AVR
• PUEDE VISUALIZARSE
• NO VISUALIZARSE OCULTA EN EL QRS
• VISUALIZARSE DESPUES DEL QRS
PRODUCIENDO UNA MELLA EN EL ST
ONDA
P
• NORMAL CON EXCEPCION SI EXISTE
PREVIAMENTE BLOQUEOS DE RAMA
• RR CONSTANTE 40-60 QRS
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ARRITMIAS
SUPRAVENTRICULARES
PLANO
AURICULAR
PLANO DE LA
UNION AV
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AURICULAR
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1)TAQUICARDIA SINUSAL
2)BRADICARDIA SINUSAL
3)ARRITMIA SINUSAL
RESPIRATORIA
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FC>100,
RESPUESTA FISIOLOGICA
AL EJERCICIO ANSIEDAD,EMOCIONES,EMBARAZO
PATOLOGICAS
FIEBRE,HIPERTIROIDISMO,ANEMIA FARMACOS
ATROPINA,ANTIASMATICOS-TEOFILINA
O
MECANISMO COMPENSADOR A FALLA CARDIACA
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FC <60
SE PRESENTA EN ATLETAS RESPUESTA VAGAL
FARMACOS (DIGITAL,BETABLOQUEADORES,)
HIPOTIROIDISMO
HIPERTENSION ENDOCRANEANA
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INSPIRACION
INHIBICION TONOVAGAL
PREDOMINA SIMPATICO
RESULTADO TAQUICARDIA
EXPIRACION
PREDOMINA PARASIMPATICO(TONO VAGAL)
RESULTADO
BRADICARDIA
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AUSENCIA DE ONDA P
1. FIBRILACION AURICULAR
REMPLAZO POR ONDAS f
2. FLUTTER AURICULAR
REEMPLAZO POR ONDAS
F
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AURICULAR
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FA es responsable del
15-20% de strokes
isquémicos
FA aumenta el riesgo de
stroke 4 a 5 veces
FA es un factor de
riesgo independiente
para la severidad y la
recurrencia del stroke
El riesgo de stroke
persiste en fibrilados
asintomáticos
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• 1909
• LA IDENTIFICO EN
HUMANOSLEWIS
• DESCRIBIO
• LAS CARACTERISTICAS
ELECTROCARDIOGRAFICASMACKENZIE
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1. ORIGEN SUPRAVENTRICULAR QRS
ANGOSTO
2. ONDA P REMPLAZADA POR ONDAS
FIBRILATORIAS “f”
3. RITMO IRREGULAR TODO RITMO
IRREGULAR SIN P ES FIBRILACION
AURICULAR
4. FRECUENCIA DE LA ONDA F ENTRE 400 A
600 POR MINUTO
5. RESPUESTA VENTRICULAR RAPIDA -
.LENTA O INTERMEDIA
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SEGÚN EL
TIEMPO DE
EVOLUCION
SEGÚN
PRESENCIA
O NO DE
PATOLOGIA
CARDIOVAS
CULAR
SEGÚN LA
FRECUEN
CIA
VENTRICU
LAR
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DE ALTA
FRECUENCIA
VENTRICULAR
> 100 LATIDOS
L/MIN
DE MODERADA
RESPUESTA
VENTRICULAR
60-100 L/MIN
DE BAJA
RESPUESTA
VENTRICULAR
<60L/MIN
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CHADS
C FALLA CARDIACA 1 PUNTO
H HIPERTENSION 1 PUNTO
A EDAD >75 AÑOS 1 PUNTO
D DIABETES 1 PUNTO
S ACV O ICT 2 PUNTOS
CON 2 O MAS PUNTOS EL PACIENTE DEBE
ESTAR ANTICOAGULADO CON WARFARINA
CON INR ENTRE 2Y 3
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ALETEO
AURICULAR
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• ARRITMIA SUPRAVENTRICULAR
• FRECUENCIA 300+-50 ONDA F (SERRUCHO)
• F INVERTIDAS EN DII-DIII-VF
• QRS ESTRECHO,REGULAR O IRREGULAR
• RELACION 2*1 o 4*1
• MASAJE CAROTIDEO VALOR DIAGNOSTICO
NO TERAPEUTICO –AUSCULTAR CAROTIDA
LADO DERECHO EN DIESTRO POR 5 A 10
SEGUNDOS
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•RITMO IRREGULAR
SIN P ES FA
•RITMO REGULAR SIN
P RITMO DE LA UNION
AV O DE LA ZONA AV
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PRIMER GRADO
CONDUCCION LENTA
•PR
>020
SEGÚNDO GRADO
CONDUCCION
INTERMITENTE
•MOBITZ 1 O
WENCKEBACH
• AUMENTO
PROGRESIVO DEL
PR QUE TERMINA
CON UNA P QUE
NO CONDUCE
•MOBITZ 2
•PR CONSTANTE
NORMAL O ALARGADO
CON UNA P
BLOQUEADA
•TIPO 2-1
•DE ALTO GRADO
TERCER GRADO O
COMPLETO
CONDUCCION AUSENTE
• DISOCIACION
AV
• PP CONSTANTE,
• RR
CONSTANTE,
• PR VARIABLE
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